Sally A. Miller
Randall C. Rowe
Richard M. Riedel
Blossom-end rot is a serious disorder of tomato, pepper, and eggplant. Growers often are distressed to notice that a dry sunken decay has developed on the blossom end (opposite the stem) of many fruit, especially the first fruit of the season. This nonparasitic disorder can be very damaging, with losses of 50% or more in some years.
On tomato and eggplant, blossom-end rot usually begins as a small water-soaked area at the blossom end of the fruit (Figure 1). This may appear while the fruit is green or during ripening. As the lesion develops, it enlarges, becomes sunken and turns black and leathery. In severe cases, it may completely cover the lower half of the fruit, becoming flat or concave. Secondary pathogens commonly invade the lesion, often resulting in complete destruction of the infected fruit. On peppers, the affected area appears tan (Figure 2), and is sometimes mistaken for sunscald, which is white. Secondary molds often colonize the affected area, resulting in a dark brown or black appearance. Blossom end rot also occurs on the sides of the pepper fruit near the blossom end.
Blossom-end rot is not caused by a parasitic organism but is a physiologic disorder associated with a low concentration of calcium in the fruit. Calcium is required in relatively large concentrations for normal cell growth. When a rapidly growing fruit is deprived of necessary calcium, the tissues break down, leaving the characteristic dry, sunken lesion at the blossom end. Blossom-end rot is induced when demand for calcium exceeds supply. This may result from low calcium levels or high amounts of competitive cations in the soil, drought stress, or excessive soil moisture fluctuations which reduce uptake and movement of calcium into the plant, or rapid, vegetative growth due to excessive nitrogen fertilization.
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